According to this alternative proposal, coronary atherosclerosis is not related to inflammation and can occur in individuals with normal circulating levels of LDL, consistent with research findings. https://t.co/DsFvGFKpuA
@vegadebates @waltoshi @realDaveFeldman If the ldl hypothesis is true in its current form, with ldl making it's way into the intima in a gradient like way, then why does it first accumulate on the outer edge of the intima away from the lumen? https://t.co/
@drpablocorral Thank you Dr. Corral. Someone presented this as an alternative hypothesis. Can you, @Drlipid @JohnKastelein @Lipoprotein and/or @DrMichaelShapir briefly respond/rebut? Would love to share your thoughts back to them. https://t.co/0PP0IXlGe9 h
@JohnnyScott234 @mikeoniron @exfatloss @jerryteixeira @KetoCarnivore @gri999999 @garytaubes @SBakerMD @ZKForTre @PeterAttiaMD ... read this paper by Subbotin, it's very accessible: https://t.co/DlCXL01S2j
@theproof @Drlipid Interested in feedback on this https://t.co/VWiEu0DuoM
@TuckerGoodrich i jumped the gun😂 https://t.co/PvJaFQ7jGp
@JohnColeman675 the way you phrased that makes me think you might be sincere. here's me taking a chance! 1) Lipoproteins invade coronary intima via neovascularization from adventitial vasa vasorum, but not from the arterial lumen https://t.co/1IlcabKb6I
@AlexJLeaf @BitcoinRadiolo1 @Lpa_Doc I don’t subscribe to that hypothesis of heart disease. I’m closer to Subbotin’s one https://t.co/ZzIGPQrti9
@CharlesMBrenner no, these are 10 times better https://t.co/Y5TqXaIUrY and https://t.co/CYjxhJk8pe
@p70S6K @deirdre_tobias these 2 papers by Subbotin really address the mechanistic evidence of the diet-heart hypothesis https://t.co/Y5TqXaIUrY and https://t.co/CYjxhJk8pe if you listen to Sniderman, Attia or Dayspring they'll all say luminal apoB infiltr
@WPM_Amar @The_Nutrivore @Tom_Babington1 I'm going with Subotin's hypothesis. https://t.co/iqPO9T4xkH
@ethanjweiss @AlastairMcA30 @Drlipid Exactly. The presence of staph/LDL isn't causal. Some other factors are causal. I think in CVD, Subbotin's theory is a very good candidate to explain the root cause of CVD. https://t.co/iqPO9T4xkH
@PeterFaberSP @NathanEqualsOne @DaveKeto @FatEmperor They’re part of the middle layer of the artery called the media. Sometimes additional vasculature forms into the intima — this is one of the early abnormalities. https://t.co/Z1ZSfUK0QH
@NathanEqualsOne 1/ Subbotin's papers seem complimentary to this https://t.co/Y5TqXaIUrY '16 https://t.co/CYjxhJk8pe '14
@Salehti @POhukainen @FatEmperor @DaveKeto In a previous series of 5 replies you've claimed to have demonstrated that in a paper from your group (still, you just infer the hydrolysis). I suppose you also discard the neovascularization from adventitial vasa
@MilesHighLife @KetoCarnivore @BarryCPearson @paleocanteen @honeybunspilsby @jen_unwin @realfoodrocksUK @PHCukorg I found it thanks! 😊 I read another paper that turned CVD hypothesis on its head: https://t.co/r9pZjU12w7 It closes the door on many theories
RT @ADreyzen: @LDLSkeptic @eatmostlyfatali @FructoseNo @ProfTimNoakes @DrAseemMalhotra I'm sure you're aware of Subbotin's work https://t.c…
@FedupfatguyK @ProfTimNoakes @jimsliwa @eatmostlyfatali @LDLSkeptic @OZmandia @FructoseNo @DrAseemMalhotra That’s at Best a grey area with every professional still blaming Cholesterol which otherwise is an essential element for healthy human life. However,
Coronary Atherosclerosis science needs to be researched further.
@LDLSkeptic @eatmostlyfatali @FructoseNo @ProfTimNoakes @DrAseemMalhotra I'm sure you're aware of Subbotin's work https://t.co/y1QAUkLjQC of all people you seem to understand it best
@POhukainen So Mr. Subbotin's hypothesis https://t.co/eQkwcrYOzI is....
@PeterAttiaMD DearPeter,thank you so much for your podcast.What is your oppinoin about this?https://t.co/JIjYjawHye
@Sadrien_GW2 @kevinnbass Because it's not ONLY LDL-c that causes atherosclerosis. https://t.co/eQkwcrYOzI
https://t.co/Y5TqXaIUrY 1 https://t.co/CYjxhJk8pe 2 You just got doubled-Subbotin'd
@DrScottMurray @FatEmperor @janvyjidak Are you talking about neovascularisation of the coronary tunica intima? https://t.co/eQkwcrYOzI
@DrScottMurray @FatEmperor @cholmoderator @Plantbiased @TuckerGoodrich @IanA_Aus @erreyedoc @AlastairMcA30 @SkepticSheep @eosull @zoeharcombe @malcolmken You implying that Ridker "cooked the books"? 🤔I'm saying there's a mechanism linking LDL with plaque.
Whether it gets in directly, or via neovascularisation, chol, fat & Ca can accumulate in media. LDL & TGs relevant! https://t.co/eQkwcrYOzI https://t.co/OZlb62LFFx
@xgmoll Tendrá razón Subbotin? https://t.co/8kGo0mdkqg Seremos nosotros los locos y Él el cuerdo? https://t.co/gwWwalHLcL
Came across similar idea a while back https://t.co/FXfrQ5SYpc Wasn't sure if fringe theory or something more Very interesting @Paddy_Barrett https://t.co/FfBUu4JE3C
Sabes cual es la hipótesis de Vladimir Subbotin, sobre el origen de la aterosclerosis.? https://t.co/hDCa6S2P91 Locura o realidad?
Neovascularization of coronary tunica intima (DIT) is the cause of coronary atherosclerosis. https://t.co/wJ65h6McwT
RT @Miguel__Prol: @puddleg @zoeharcombe @FatEmperor We know for sure he's not for the Subbotin's hypothesis https://t.co/Wk8QWiV8LT
@puddleg @zoeharcombe @FatEmperor We know for sure he's not for the Subbotin's hypothesis https://t.co/Wk8QWiV8LT
@PeterLu9 My post contains a *lot* of studies showing causation mechanism. CHD is also LDL-C mediated, according to https://t.co/zSR2jyqBQW
@PauliOhukainen Does this make any sense to you? Above my pay grade. http://t.co/PrDD1lJIPE http://t.co/4lMRuekduu Plain english?
Great Hypothesis paper on likely primary cause of Atherosclerosis - it ain't the LDL/fat! http://t.co/C3ngtHyqvD http://t.co/AC1CGdaXso
@Gearoidmuar @ianlane88 @Drlipid Vladimir is frisky - Gearoid a chara, you're a histopathologist - céard é do mheas? http://t.co/s4csDFcoKu
If this study is accurate, statins will only slow your progress towards a heart attack: http://t.co/c1zr0Ft2Wd
@MikkiWilliden @ONENewsNZ for example, hypothesised mechanism http://t.co/wTzJbScLYI
Great Hypothesis paper on likely primary cause of Atherosclerosis - it ain't the LDL/fat! http://t.co/C3ngtHyqvD http://t.co/AC1CGdaXso
Great Hypothesis paper on likely primary cause of Atherosclerosis - it ain't the LDL/fat! http://t.co/C3ngtHyqvD http://t.co/AC1CGdaXso
A new theory of coronary artery atherosclerotic pathogenesis: neovascularisation of tunica intima (not LDL excess) http://t.co/yGJUSMaBvy
Great Hypothesis paper on likely primary cause of Atherosclerosis - it ain't the LDL/fat! http://t.co/C3ngtHyqvD http://t.co/AC1CGdaXso
Great Hypothesis paper on likely primary cause of Atherosclerosis - it ain't the LDL/fat! http://t.co/C3ngtHyqvD http://t.co/AC1CGdaXso
Great Hypothesis paper on likely primary cause of Atherosclerosis - it ain't the LDL/fat! http://t.co/C3ngtHyqvD http://t.co/AC1CGdaXso
Great Hypothesis paper on likely primary cause of Atherosclerosis - it ain't the LDL/fat! http://t.co/C3ngtHyqvD http://t.co/AC1CGdaXso
Great Hypothesis paper on likely primary cause of Atherosclerosis - it ain't the LDL/fat! http://t.co/C3ngtHyqvD http://t.co/AC1CGdaXso
@wyadvd @FatEmperor @cvictorg Any comments on http://t.co/zSR2jyr9Gu ? ↑ LDL-P → ↑ LDL entry to DIT. LDL-P ∝ LDL-C. LDL-P ∝ 1/(LDL-Size)^3
@Drlipid What do you think of Subbotin's "Neovascularisation of coronary tunica intima (DIT)" hypothesis http://t.co/tyk6WM92Eo ?
Neovascularization of coronary tunica intima (DIT) is the cause of coronary atherosclerosis. Lipoproteins invade... http://t.co/ryNwcjeKiO
Neovascularization of coronary tunica intima (DIT) is the cause of coronary atherosclerosis. Lipoproteins invade... http://t.co/ryNwcjeKiO
Neovascularization of coronary tunica intima (DIT) is the cause of coronary atherosclerosis. Lipoproteins invade... http://t.co/ryNwcjeKiO